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Therefore, it is reasonable using supplementation of liposomal glutathione, instead of the N-acetylcysteine or bonded cysteine utilized as precursors for GSH cell synthesis, since patients with deficient levels of glutamate-cysteine ligase and GSH synthetase will not be able to use N-acetylcysteine or bonded cysteine as substrates to synthesize their own GSH

Meanwhile, RSMC depletion did not change S phase progression significantly, suggesting that the reduced Sororincohesin interaction is unlikely due to the cell cycle delay (Fig